Study group discussion: Diabetes

I’ve noticed any thing with my “swedish” medical main division …. It always give glucose values in ( mmmol/l ) and not ( mg/dl )

fP-Glucose > 7,0 mmol/l
How a great quantity mg/dl is that ?

The employment factor is   mg/dl= 18 x mmol/l
So 7mmol/l is 126mg/dl
I’m truly majoring in pharmacology 🙂 we had a chance of those ^^

HbA1c < 52 mmol/l = 6 %
For diabetes exemplar 1
Hemoglobin A1c level should exist held at 6%
I’m version about diabetes type 1 treatment
And it’s written in this place HbA1c level should be held < 52 mmol/mol
equals to 6%

*a change of religion table was posted on the assemblage by someone which sorted this awkward*

That might be possible but because of the diagnosis it is >6.5

You slip on’t use A1c for diagnosis. It is used barely for evaluating control over the in conclusion few months!

But now if it’s >6.5 he is diagnosed diabetic.

Umm however.. Why would you use such a test when there are better screening tests?

Ummm its faction of one of the criteria on the side of diagnosis!! Though getting fasting samples and tot~y are easy but you have to complete the patient fast right!! In this the same benefit would be take the samples anytime!

Oh yep.. Makes brains!

A diabetic patient is undergoing difference angiography for some reason. He was asked to suppress his oral hypoglycemic, metformin before the proceeding. Why?

Lactic acidosis.
Renal failure choose worsen it.
Contrast leads to renal injury.

Correct! You are concerned about lactic acidosis. Even cursory renal failure due to the stain will precipitate lactic acidosis!

A diabetic patient controlled on Insulin develops fainting episodes. On investigations, he was build to have an elevated creatinine. What is the mechanical construction for the hypoglycemia?

Insulin excreted renally?

Yes, the half life of insulin is increased in renal failure!

A foster presents with hypoglycemia. Her insulin levels are up further C peptide levels are low. Diagnosis?

Taking exogenous insulin.

Alright. You face the patient and goes into counselling.

Now the sort patient comes with hypoglycemia but this time her insulin and C peptide levels, one as well as the other are elevated!
What happened this time?

Using sulphonylureas
Oral diabetic drugs – Glipizide, glimipiride, glyburide

Correct! How command you prove it?

proInsulin levels? Just guessing put on’t know that!!


What’s u/a?

U/A is shortening for urine analysis

Correct! Urine beneficial to Sulfonylureas

If the urine test came negative, the sort of could it be?


And he scores again!

Wonderful questions!!

That’ll be all for today!

Thank you during the great questions IkaN.

Never reasoning questions would be this much sport!

*After which someone else asked us a few review questions based on what he had closely examined! *

Which antidiabetic drug can lead to SIADH?


I would take that!! All sulphonylureas be at the head of to SIADH.

Ok one more!! How would you attack severity of diabetic ketoacidosis?

*since no one could guess, we were given hints!*

Let me clown in this way.. Which electrolyte would you Check in serum to way severity of DKA?

If you had to bridle just 1.



Why bicarbonate?

Because it is acidosis

Yes, you are equitable!!

Low bicarbonate would lead to the sort of? This one entity is very significant in DKA management!  If this is corrected sufferer is well and good!! Some debate in the cations and anions!! What’s that called?

Anion interstice

Yes, it is!! Finally!!

This anion rift is very important.

What the regular value?

10 to 15

Does this break increase or decrease?


ABG would own is about acid base imbalance.

How does neuropathy occur in diabetes? What’s the mechanical construction?

Occlusion of small venules?

Damage to autonomic NS..?

Microvascular occlusion?

Yes, nerves themselves possess a supply of blood vessels. Diabetes indemnity these small blood vessels, thus decreases furnish to nerves!!

You all were amend!!

I’ve read somewhere that sorbitol deposits too damage nerves?

Lens! It causes waterfall.

Alright guys!! That’s it!! I be possible to’t remember any more!!

Any mnemonics?

Here are every part of the study links! into ~s-discussion-pharmacological_15.html into ~s-discussion-type-3-diabetes.html

Extremely moo blood pressure can causeshock and in the end, departure.

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